Lack of microRNA-155 ameliorates renal fibrosis by targeting PDE3A/TGF-β1/Smad signaling in mice with obstructive nephropathy-Reference-Cited by-同舟云学术

Lack of microRNA-155 ameliorates renal fibrosis by targeting PDE3A/TGF-β1/Smad signaling in mice with obstructive nephropathy

Published:2018-09-14 Issue:11 Volume:42 Page:1523-1532
ISSN:1065-6995
Container-title:Cell Biology International
language:en
Short-container-title:Cell Biol Int

Author:

Xi Weiwei¹,Zhao Xuming¹,Wu Meijun²,Jia Wenjuan¹,Li Hua¹

Affiliation:

1. Department of Nephrology; Zhejiang University Medical College Affiliated Sir Run Run Shaw Hospital; Qingchun Road 3rd Hangzhou 310016 Zhejiang Province China

2. Department of Comprehensive Health Care; The First People's Hospital of Hangzhou; Hangzhou 310016 Zhejiang Province China

Publisher

Wiley

Subject

Cell Biology,General Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/cbin.11038/fullpdf

Reference38 articles.

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2. MicroRNA-155 functions as a negative regulator of RhoA signaling in TGF-beta-induced endothelial to mesenchymal transition;Bijkerk;Microrna,2012

3. MiR-155 in the progression of lung fibrosis in systemic sclerosis;Christmann;Arthritis Res Ther,2016

4. MiR-192 mediates TGF-beta/Smad3-driven renal fibrosis;Chung;J Am Soc Nephrol,2010

5. MicroRNAs in renal fibrosis;Chung;Front Physiol,2015

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