Tanshinone IIA alleviates acute ethanol‐induced myocardial apoptosis mainly through inhibiting the expression of PDCD4 and activating the PI3K /Akt pathway-Reference-Cited by-同舟云学术

Tanshinone IIA alleviates acute ethanol‐induced myocardial apoptosis mainly through inhibiting the expression of PDCD4 and activating the PI3K /Akt pathway

Published:2021-06-24 Issue:8 Volume:35 Page:4309-4323
ISSN:0951-418X
Container-title:Phytotherapy Research
language:en
Short-container-title:Phytotherapy Research

Author:

Deng Hanyu¹^ORCID,Yu Bo¹,Li Yang¹

Affiliation:

1. Department of Cardiology The First Affiliated Hospital of China Medical University Shenyang China

Publisher

Wiley

Subject

Pharmacology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ptr.7102

Reference48 articles.

1. Tanshinone IIA ameliorates oxaliplatin‐induced neurotoxicity via mitochondrial protection and autophagy promotion;Cheng W.;American Journal of Translational Research,2019

2. MicroRNA-21 protects against the H2O2-induced injury on cardiac myocytes via its target gene PDCD4

3. NO66 overexpression rescues ethanol-induced cell apoptosis in human AC16 cardiomyocytes by suppressing PTEN and activating the PI3K/Akt signaling

4. MiR-21 attenuates apoptosis-triggered by amyloid-β via modulating PDCD4/ PI3K/AKT/GSK-3β pathway in SH-SY5Y cells

5. The Effects of Ethanol on the Heart: Alcoholic Cardiomyopathy

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