Overexpression of IGF-IIRα regulates cardiac remodeling and aggravates high salt induced apoptosis and fibrosis in transgenic rats-Reference-Cited by-全球学者库

Overexpression of IGF-IIRα regulates cardiac remodeling and aggravates high salt induced apoptosis and fibrosis in transgenic rats

Published:2018-11-18 Issue:2 Volume:34 Page:210-218
ISSN:1520-4081
Container-title:Environmental Toxicology
language:en
Short-container-title:Environmental Toxicology

Author:

Chang Ruey-Lin¹,Nithiyanantham Srinivasan²,Kuo Wei-Wen³,Pai Pei-Ying⁴,Chang Tung-Ti⁵,Lai Chao-Hung⁶,Chen Ray-Jade⁷,Vijaya Padma Viswanadha⁸,Huang Chih-Yang⁹,Huang Chih-Yang²⁵¹⁰^ORCID

Affiliation:

1. School of Post-Baccalaureate Chinese Medicine; College of Chinese Medicine, China Medical University; Taichung Taiwan

2. Graduate Institute of Basic Medical Science; China Medical University; Taichung Taiwan

3. Department of Biological Science and Technology; China Medical University; Taichung Taiwan

4. Division of Cardiology; China Medical University Hospital; Taichung Taiwan

5. School of Chinese Medicine; China Medical University; Taichung Taiwan

6. Department of Internal Medicine, Division of Cardiology; Armed Forces Taichung General Hospital; Taichung Taiwan

7. Department of Surgery, School of Medicine, College of Medicine; Taipei Medical University; Taipei Taiwan

8. Department of Biotechnology; Bharathiar University; Coimbatore India

9. Translation Research Core; China Medical University Hospital, China Medical University; Taichung Taiwan

10. Department of Biological Science and Technology; Asia University; Taichung Taiwan

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Management, Monitoring, Policy and Law,Toxicology,General Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/tox.22676/fullpdf

Reference38 articles.

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4. High salt intake damages the heart through activation of cardiac (pro) renin receptors even at an early stage of hypertension;Hayakawa;Plos One,2015

5. High-salt diet and hypertension: focus on the renin-angiotensin system;Drenjancevic-Peric;Kidney Blood Press Res,2011

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