Delay in expression of a mammary tumor provirus is responsible for defective clonal deletion during postnatal period
Author:
Publisher
Wiley
Subject
Immunology,Immunology and Allergy
Reference27 articles.
1. T cell tolerance by clonal elimination in the thymus
2. Self-tolerance eliminates T cells specific for Mls-modified products of the major histocompatibility complex
3. T-cell receptor Vβ use predicts reactivity and tolerance to Mlsa- encoded antigens
4. A superantigen encoded in the open reading frame of the 3′ long terminal repeat of mouse mammary tumour virus
5. Clonal deletion of V β814-bearing T cells in mice transgenic for mammary tumour virus
Cited by 6 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Expression of Mouse Mammary Tumor Virus Superantigen Accelerates Tumorigenicity of Myeloma Cells;Journal of Virology;2000-09-15
2. Failure to remove autoreactive Vβ6+ T cells in Mls-1a newborn mice attributed to the delayed development of B cells in the thymus;Immunology;2000-08
3. Mouse Mammary Tumor Virus: Immunological Interplays between Virus and Host **This article was accepted for publication on 1 October 1996.;Advances in Immunology;1997
4. A novel V beta 2-specific endogenous mouse mammary tumor virus which is capable of producing a milk-borne exogenous virus;Journal of Virology;1995-11
5. Quantitation of endogenous mouse mammary tumor virus superantigen expression by lymphocyte subsets;European Journal of Immunology;1995-09
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