M2 macrophage accumulation contributes to pulmonary fibrosis, vascular dilatation, and hypoxemia in rat hepatopulmonary syndrome
Author:
Affiliation:
1. Department of Anesthesia The Second Affiliated Hospital of Chongqing Medical University Chongqing China
2. Department of Anesthesia, Southwest Hospital Army Medical University (The Third Military Medical University) Chongqing China
Funder
National Natural Science Foundation of China
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.30420
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1. Arterial Changes in the Lungs in Cirrhosis of the Liver — Lung Spider Nevi
2. Caspase-3 inhibition prevents the development of hepatopulmonary syndrome in common bile duct ligation rats by alleviating pulmonary injury
3. A self‐organized actomyosin drives multiple intercellular junction disruption and directly promotes neutrophil recruitment in lipopolysaccharide‐induced acute lung injury
4. Dexmedetomidine Attenuates Bilirubin-Induced Lung Alveolar Epithelial Cell Death In Vitro and In Vivo*
5. Modulating Macrophage Polarization through CCR2 Inhibition and Multivalent Engagement
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