Low‐dose TNF augments fracture healing in normal and osteoporotic bone by up‐regulating the innate immune response-Reference-Cited by-同舟云学术

Low‐dose TNF augments fracture healing in normal and osteoporotic bone by up‐regulating the innate immune response

Published:2015-03-14 Issue:5 Volume:7 Page:547-561
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Chan James K¹,Glass Graeme E¹,Ersek Adel¹,Freidin Andrew¹,Williams Garry A¹,Gowers Kate²,Espirito Santo Ana I¹,Jeffery Rosemary³,Otto William R³,Poulsom Richard³,Feldmann Marc¹,Rankin Sara M²,Horwood Nicole J¹,Nanchahal Jagdeep¹

Affiliation:

1. Kennedy Institute of Rheumatology University of Oxford Oxford UK

2. National Heart and Lung Institute Imperial College London London UK

3. Histopathology Laboratory and In Situ Hybridisation Service Cancer Research UK – London Research Institute London UK

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201404487

Reference101 articles.

1. High Levels of Tumor Necrosis Factor-α Contribute to Accelerated Loss of Cartilage in Diabetic Fracture Healing

2. Osteal macrophages promote in vivo intramembranous bone healing in a mouse tibial injury model

3. Effect of Nonsteroidal Antiinflammatory Drugs on Fracture Healing

4. Peptide fragment 29–40 of amino acid sequence of monocyte chemoattractant protein-1 (MCP-1) stimulates monocyte migration in vivo and facilitates wound healing

5. Update on Bone Anabolics in Osteoporosis Treatment: Rationale, Current Status, and Perspectives

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