FBXL 4 deficiency increases mitochondrial removal by autophagy-Reference-Cited by-同舟云学术

FBXL 4 deficiency increases mitochondrial removal by autophagy

Published:2020-06-11 Issue:7 Volume:12 Page:
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Alsina David¹²,Lytovchenko Oleksandr¹²,Schab Aleksandra¹,Atanassov Ilian³^ORCID,Schober Florian A²⁴,Jiang Min⁵,Koolmeister Camilla¹²,Wedell Anna⁴⁶,Taylor Robert W⁷^ORCID,Wredenberg Anna¹²⁶,Larsson Nils‐Göran¹²⁶^ORCID

Affiliation:

1. Department of Medical Biochemistry and Biophysics Karolinska Institutet Stockholm Sweden

2. Max Planck Institute Biology of Ageing ‐ Karolinska Institutet Laboratory Karolinska Institutet Stockholm Sweden

3. Proteomics Core Facility Max Planck Institute for Biology of Ageing Cologne Germnay

4. Department of Molecular Medicine and Surgery Karolinska Institutet Stockholm Sweden

5. Key Laboratory of Growth Regulation and Translation Research of Zhejiang Province School of Life Sciences, Westlake University Hangzhou China

6. Centre for Inherited Metabolic Diseases Karolinska University Hospital Stockholm Sweden

7. Wellcome Centre for Mitochondrial Research, Translational and Clinical Research Institute Newcastle University Newcastle upon Tyne UK

Funder

Svenska Forskningsrådet Formas

Knut och Alice Wallenbergs Stiftelse

H2020 European Research Council

Cancerfonden

Sveriges Regering

UK Research and Innovation

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201911659

Reference53 articles.

1. Loss of iron triggers PINK1/Parkin‐independent mitophagy

2. Detailed Biochemical and Bioenergetic Characterization of FBXL4-Related Encephalomyopathic Mitochondrial DNA Depletion

3. Regulation of autophagy by the Rab GTPase network

4. Mutations in FBXL4 Cause Mitochondrial Encephalopathy and a Disorder of Mitochondrial DNA Maintenance

5. Retro-translocation of mitochondrial intermembrane space proteins

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