Targeting macrophage Histone deacetylase 3 stabilizes atherosclerotic lesions-Reference-Cited by-同舟云学术

Targeting macrophage Histone deacetylase 3 stabilizes atherosclerotic lesions

Published:2014-07-09 Issue:9 Volume:6 Page:1124-1132
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Hoeksema Marten A¹,Gijbels Marion JJ¹²³,Van den Bossche Jan¹,Velden Saskia¹,Sijm Ayestha¹,Neele Annette E¹,Seijkens Tom¹,Stöger J Lauran¹,Meiler Svenja¹,Boshuizen Marieke CS¹,Dallinga‐Thie Geesje M⁴,Levels Johannes HM⁴,Boon Louis⁵,Mullican Shannon E⁶,Spann Nathanael J⁷,Cleutjens Jack P²,Glass Chris K⁷,Lazar Mitchell A⁶,Vries Carlie JM¹,Biessen Erik AL²,Daemen Mat JAP⁸,Lutgens Esther¹⁹,Winther Menno PJ¹

Affiliation:

1. Department of Medical Biochemistry, Experimental Vascular Biology Academic Medical Center University of Amsterdam Amsterdam The Netherlands

2. Department of Pathology Maastricht University Maastricht The Netherlands

3. Department of Molecular Genetics Maastricht University Maastricht The Netherlands

4. Department of Vascular and Experimental Vascular Medicine Academic Medical Center University of Amsterdam Amsterdam The Netherlands

5. Bioceros BV Utrecht The Netherlands

6. Division of Endocrinology, Diabetes and Metabolism Department of Medicine Institute for Diabetes, Obesity and Metabolism Perelman School of Medicine University of Pennsylvania Philadelphia USA

7. Department of Cellular and Molecular Medicine University of California San Diego CA USA

8. Department of Pathology Academic Medical Center University of Amsterdam Amsterdam The Netherlands

9. Institute for Cardiovascular Prevention (IPEK) Ludwig Maximilian's University Munich Germany

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201404170

Reference32 articles.

1. Epigenetic protein families: a new frontier for drug discovery

2. The Bcl6-SMRT/NCoR Cistrome Represses Inflammation to Attenuate Atherosclerosis

3. Wound Macrophages as Key Regulators of Repair

4. Requirement for the histone deacetylase Hdac3 for the inflammatory gene expression program in macrophages

5. The phenotype of murine wound macrophages

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