The IKK‐related kinase TBK1 activates mTORC1 directly in response to growth factors and innate immune agonists-Reference-Cited by-同舟云学术

The IKK‐related kinase TBK1 activates mTORC1 directly in response to growth factors and innate immune agonists

Published:2017-11-17 Issue:1 Volume:37 Page:19-38
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Bodur Cagri¹,Kazyken Dubek¹,Huang Kezhen¹,Ekim Ustunel Bilgen¹,Siroky Kate A¹,Tooley Aaron Seth¹,Gonzalez Ian E¹,Foley Daniel H¹,Acosta‐Jaquez Hugo A¹,Barnes Tammy M²,Steinl Gabrielle K²,Cho Kae‐Won³,Lumeng Carey N³,Riddle Steven M⁴,Myers Martin G²,Fingar Diane C¹^ORCID

Affiliation:

1. Department of Cell and Developmental Biology University of Michigan Medical School Ann Arbor MI USA

2. Department of Internal Medicine University of Michigan Medical School Ann Arbor MI USA

3. Department of Pediatrics and Communicable Diseases University of Michigan Medical School Ann Arbor MI USA

4. Illumina Madison WI USA

Funder

National Institute of Diabetes and Digestive and Kidney Diseases

American Diabetes Association

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.201696164

Reference90 articles.

1. Site-Specific mTOR Phosphorylation Promotes mTORC1-Mediated Signaling and Cell Growth

2. Vesicular stomatitis virus oncolysis is potentiated by impairing mTORC1-dependent type I IFN production

3. New Insights into mTOR Signaling: mTORC2 and Beyond

4. Systematic RNA interference reveals that oncogenic KRAS-driven cancers require TBK1

5. Integrative Genomic Approaches Identify IKBKE as a Breast Cancer Oncogene

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