Cyclin A triggers Mitosis either via the Greatwall kinase pathway or Cyclin B-Reference-Cited by-同舟云学术

Cyclin A triggers Mitosis either via the Greatwall kinase pathway or Cyclin B

Published:2020-04-30 Issue:11 Volume:39 Page:
ISSN:0261-4189
Container-title:The EMBO Journal
language:en
Short-container-title:EMBO J

Author:

Hégarat Nadia¹,Crncec Adrijana¹^ORCID,Suarez Peredo Rodriguez Maria F¹^ORCID,Echegaray Iturra Fabio¹^ORCID,Gu Yan¹,Busby Oliver¹,Lang Paul F²,Barr Alexis R³⁴^ORCID,Bakal Chris⁵,Kanemaki Masato T⁶⁷,Lamond Angus I⁸^ORCID,Novak Bela²^ORCID,Ly Tony⁹^ORCID,Hochegger Helfrid¹^ORCID

Affiliation:

1. Genome Damage and Stability Centre School of Life Sciences University of Sussex Brighton UK

2. Department of Biochemistry University of Oxford Oxford UK

3. MRC London Institute of Medical Science Imperial College London UK

4. Institute of Clinical Sciences Faculty of Medicine Imperial College London UK

5. Institute for Cancer Research Chester Beatty Laboratories London UK

6. National Institute of Genetics Research Organization of Information and Systems (ROIS) Mishima Japan

7. Department of Genetics SOKENDAI (The Graduate University of Advanced Studies) Mishima Japan

8. Centre for Gene Regulation and Expression School of Life Sciences University of Dundee Dundee UK

9. Wellcome Trust Centre for Cell Biology University of Edinburgh Edinburgh UK

Funder

Cancer Research UK

Wellcome Trust

Biotechnology and Biological Sciences Research Council

Wolfson Foundation

Publisher

EMBO

Subject

General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,Molecular Biology,General Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embj.2020104419

Reference58 articles.

1. MAD1-dependent recruitment of CDK1-CCNB1 to kinetochores promotes spindle checkpoint signaling

2. Greatwall is essential to prevent mitotic collapse after nuclear envelope breakdown in mammals

3. Covalent capture of kinase-specific phosphopeptides reveals Cdk1-cyclin B substrates

4. A Quantitative Model for Ordered Cdk Substrate Dephosphorylation during Mitotic Exit

5. Cyclin B2-null mice develop normally and are fertile whereas cyclin B1-null mice die in utero

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