Advances in the structural mechanism of TRPC channels
Author:
Publisher
Science China Press., Co. Ltd.
Subject
Multidisciplinary
Link
https://engine.scichina.com/doi/pdf/39EC0E6138D6462CA26154ECCC198CFB
Reference43 articles.
1. Inrig J K, Califf R M, Tasneem A. The landscape of clinical trials in nephrology: A systematic review of Clinicaltrials.gov. Am J Kidney Dis, 2014, 63: 771-780.
2. Reiser J, Polu K R, Möller C C. TRPC6 is a glomerular slit diaphragm-associated channel required for normal renal function. Nat Genet, 2005, 37: 739-744.
3. Winn M P, Conlon P J, Lynn K L. A mutation in the TRPC6 cation channel causes familial focal segmental glomerulosclerosis. Science, 2005, 308: 1801-1804.
4. Riehle M, Büscher A K, Gohlke B O. TRPC6 G757D loss-of-function mutation associates with FSGS. J Am Soc Nephrol, 2016, 27: 2771-2783.
5. Liu Z, Zhang H, Zhao S. Novel gain-of-function mutation of TRPC6 Q134P contributes to late onset focal segmental glomerulosclerosis in a Chinese pedigree. Nephrology, 2021, 26: 1018-1025.
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