Voltage-gated calcium channels - from basic mechanisms to disease
Author:
Affiliation:
1. Department of Pharmacology and Toxicology; Centre for Molecular Biosciences; University of Innsbruck; Innrain 80/82, 6020 Innsbruck Austria
Funder
Austrian Science Fund
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1113/JP272619/fullpdf
Reference36 articles.
1. A single amino acid deletion (ΔF1502) in the S6 segment of Cav2.1 domain III associated with congenital ataxia increases channel activity and promotes Ca2+ influx;Bahamonde;PLoS ONE,2015
2. Loss of Cav1.3 (CACNA1D) function in a human channelopathy with bradycardia and congenital deafness;Baig;Nat Neurosci,2011
3. Transport and metabolism of calcium ions in nerve;Baker;Prog Biophys Mol Biol,1972
4. Role of voltage-gated calcium channels in the regulation of aldosterone production from zona glomerulosa cells of the adrenal cortex;Barrett;J Physiol,2016
5. Channelopathies and dendritic dysfunction in Fragile X syndrome;Brager;Brain Res Bull,2014
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