p53: the tumour suppressor turns mitochondrial regulator
Author:
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1113/jphysiol.2013.259481/fullpdf
Reference5 articles.
1. Matched work high-intensity interval and continuous running induce similar increases in PGC-1α mRNA, AMPK, p38, and p53 phosphorylation in human skeletal muscle
2. Reduced carbohydrate availability enhances exercise-induced p53 signaling in human skeletal muscle: implications for mitochondrial biogenesis
3. Role of p53 in mitochondrial biogenesis and apoptosis in skeletal muscle
4. Acute exercise induces tumour suppressor protein p53 translocation to the mitochondria and promotes a p53-Tfam-mitochondrial DNA complex in skeletal muscle
5. p53 in health and disease
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1. Cellular and molecular mechanisms of oroxylin A in cancer therapy: Recent advances;European Journal of Pharmacology;2024-04
2. Down‐regulation of TFAM increases the sensitivity of tumour cells to radiation via p53/TIGAR signalling pathway;Journal of Cellular and Molecular Medicine;2019-05-06
3. Oroxylin A modulates mitochondrial function and apoptosis in human colon cancer cells by inducing mitochondrial translocation of wild-type p53;Oncotarget;2016-03-05
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