Excess glucose induces hypoxia-inducible factor-1α in pancreatic cancer cells and stimulates glucose metabolism and cell migration
Author:
Publisher
Informa UK Limited
Subject
Cancer Research,Pharmacology,Oncology,Molecular Medicine
Link
http://www.tandfonline.com/doi/pdf/10.4161/cbt.23786
Reference25 articles.
1. A nuclear factor induced by hypoxia via de novo protein synthesis binds to the human erythropoietin gene enhancer at a site required for transcriptional activation.
2. Hypoxia-inducible Factor 1α (HIF-1α) Protein Is Rapidly Degraded by the Ubiquitin-Proteasome System under Normoxic Conditions
3. Synergistic up-regulation of Hexokinase-2, glucose transporters and angiogenic factors in pancreatic cancer cells by glucose deprivation and hypoxia
4. HIF-1 mediates adaptation to hypoxia by actively downregulating mitochondrial oxygen consumption
5. HIF-1 prevents the overproduction of mitochondrial ROS after cytokine stimulation through induction of PDK-1
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