ASO Author Reflections: A Possible New Etiology of Thymoma-Associated Myasthenia Gravis—Human Parvovirus B19
Author:
Publisher
Springer Science and Business Media LLC
Subject
Oncology,Surgery
Link
https://link.springer.com/content/pdf/10.1245/s10434-022-12958-3.pdf
Reference5 articles.
1. Leite MI, Jones M, Ströbel P, et al. Myasthenia gravis thymus: complement vulnerability of epithelial and myoid cells, complement attack on them, and correlations with autoantibody status. Am J Pathol. 2007;171:893–905.
2. Cavalcante P, Bernasconi P, Mantegazza R. Autoimmune mechanisms in myasthenia gravis. Curr Opin Neurol. 2012;25:621–9.
3. Lefeuvre CM, Payet CA, Fayet OM, et al. Risk factors associated with myasthenia gravis in thymoma patients: The potential role of thymic germinal centers. J Autoimmun. 2020;106:102337.
4. Gong L, Li Y, Li X, et al. Detection of human parvovirus B19 infection in the thymus of patients with thymic hyperplasia-associated myasthenia gravis. Clin Microbiol Infect. 2019;25:109.e7-e12.
5. Gong L, Tian J, Zhang Y, et al. Gaosheng Huang. Human parvovirus B19 may be a risk factor in myasthenia gravis with thymoma. Ann Surg Oncol. 2022. https://doi.org/10.1245/s10434-022-12936-9
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