p62 functions as an oncogene in colorectal cancer through inhibiting apoptosis and promoting cell proliferation by interacting with the vitamin D receptor-Reference-Cited by-同舟云学术

p62 functions as an oncogene in colorectal cancer through inhibiting apoptosis and promoting cell proliferation by interacting with the vitamin D receptor

Published:2019-02-22 Issue:3 Volume:52 Page:e12585
ISSN:0960-7722
Container-title:Cell Proliferation
language:en
Short-container-title:Cell Prolif

Author:

Zhang Jing¹,Yang Suzhen¹,Xu Bing²^ORCID,Wang Ting¹,Zheng Ying¹,Liu Fei³,Ren Fenggang⁴,Jiang Jiong¹,Shi Haitao¹,Zou Baicang¹,Lu Xiaolan⁵,Lu Shemin⁶,Dong Lei¹^ORCID

Affiliation:

1. Department of Digestive Disease and Gastrointestinal Motility Research Room The Second Affiliated Hospital of Xi’an Jiaotong University Xi’an China

2. Department of Digestive Disease and Gastrointestinal Motility Research Room Xi’an Jiaotong University Xi’an China

3. Clinical Research Center of Shanxi Province for Dental and Maxillofacial Diseases, College of Stomatology Xi'an Jiaotong University Xi’an China

4. National Local Joint Engineering Research Center for Precision Surgery and Regenerative Medicine Xi’an Jiaotong University Xi’an China

5. Department of Digestive Disease Shanghai Pudong Hospital, Fudan Affiliated Pudong Medical Center Shanghai China

6. Department of Biochemistry and Molecular Biology, School of Basic Medical Sciences Xi'an Jiaotong University Health Science Center Xi’an China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/cpr.12585

Reference39 articles.

1. Colorectal cancer: From prevention to personalized medicine

2. Global patterns and trends in colorectal cancer incidence and mortality

3. Autophagy protein p62/SQSTM1 is involved in HAMLET-induced cell death by modulating apotosis in U87MG cells

4. Metabolic Control of Autophagy

5. Autophagy at the crossroads of catabolism and anabolism

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