Purine nucleoside phosphorylase activity and expression are upregulated in sites affected by periodontal disease
Author:
Publisher
Wiley
Subject
Periodontics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1600-0765.2010.01282.x/fullpdf
Reference27 articles.
1. Interference with immune-cell-mediated bone resorption in periodontal disease;Han;Periodontol 2000,2007
2. Bacterial-responsive B lymphocytes induce periodontal bone resorption;Han;J Immunol,2006
3. Gamma interferon positively modulates Actinobacillus actinomycetemcomitans-specific RANKL+ CD4 + Th-cell-mediated alveolar bone destruction in vivo;Teng;Infect Immun,2005
4. Requirement of B7 costimulation for Th1-mediated inflammatory bone resorption in experimental periodontal disease;Kawai;J Immunol,2000
5. B and T lymphocytes are the primary sources of RANKL in the bone resorptive lesion of periodontal disease;Kawai;Am J Pathol,2006
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