Chronic tissue damage: a common pathomechanism of genodermatoses
Author:
Affiliation:
1. Faculty of Medicine Medical Center‐University of Freiburg Freiburg Germany
Funder
Debra International
Publisher
Wiley
Subject
Dermatology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/bjd.18104
Reference8 articles.
1. Fibroblast activation and abnormal extracellular matrix remodelling as common hallmarks in three cancer‐prone genodermatoses
2. Epidermolysis bullosa: Molecular pathology of connective tissue components in the cutaneous basement membrane zone
3. Integrin activation by talin, kindlin and mechanical forces
4. XPC is an RNA polymerase II cofactor recruiting ATAC to promoters by interacting with E2F1
5. Global remodelling of cellular microenvironment due to loss of collagen VII
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1. Unveiling the value of C‐reactive protein as a severity biomarker and the IL4/IL13 pathway as a therapeutic target in recessive dystrophic epidermolysis bullosa: A multiparametric cross‐sectional study;Experimental Dermatology;2024-07-29
2. Consensus reclassification of inherited epidermolysis bullosa and other disorders with skin fragility;British Journal of Dermatology;2020-03-11
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