Dendritic pathology in mental retardation: from molecular genetics to neurobiology-Reference-Cited by-同舟云学术

Dendritic pathology in mental retardation: from molecular genetics to neurobiology

Published:2006-06 Issue: Volume:5 Page:48-60
ISSN:1601-1848
Container-title:Genes, Brain and Behavior
language:en
Short-container-title:

Author:

Dierssen M.,Ramakers G. J. A.

Publisher

Wiley

Subject

Behavioral Neuroscience,Neurology,Genetics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1601-183X.2006.00224.x/fullpdf

Reference130 articles.

1. Down syndrome - a gene dosage disease caused by trisomy of genes within a small segment of the long arm of chromosome 21, exemplified by the study of effects from the superoxide-dismutase type 1 (SOD-1) Gene;Anneren;APMIS Suppl,1993

2. Chromosome 21 and Down syndrome: from genomics to pathophysiology;Antonarakis;Nat Rev Genet,2004

3. Actin-binding protein, drebrin, accumulates in submembranous regions in parallel with neuronal differentiation;Asada;J Neurosci Res,1994

4. Abnormal pattern of cerebral glucose metabolic rates involving language areas in young adults with Down syndrome;Azari;Brain Lang,1994

5. Receptive fields of cricket giant interneurones are related to their dendritic structure;Bacon;J Physiol,1984

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