Obesity increases allergic airway inflammation that can be successfully treated by oral tolerance-Reference-Cited by-同舟云学术

Obesity increases allergic airway inflammation that can be successfully treated by oral tolerance

Published:2023-12-27 Issue:2 Volume:79 Page:529-533
ISSN:0105-4538
Container-title:Allergy
language:en
Short-container-title:Allergy

Author:

Geissler Nora¹^ORCID,Orola Maria¹,Alinaghi Masoumeh²^ORCID,Nardo Alexander³^ORCID,Stulnig Thomas M.³⁴^ORCID,Séneca Joana⁵⁶^ORCID,Schmid Anna¹^ORCID,Korb Elke¹,Svoboda Tatjana²,Garner‐Spitzer Erika¹^ORCID,Kundi Michael¹^ORCID,Ehling‐Schulz Monika²^ORCID,Schabussova Irma¹^ORCID,Inic‐Kanada Aleksandra¹^ORCID,Wiedermann Ursula¹^ORCID

Affiliation:

1. Institute of Specific Prophylaxis and Tropical Medicine, Center for Pathophysiology, Infectiology and Immunology Medical University of Vienna Vienna Austria

2. Functional Microbiology, Institute of Microbiology, Department of Pathobiology University of Veterinary Medicine Vienna Austria

3. Clinical Division of Endocrinology & Metabolism, Department of Medicine III Medical University of Vienna Vienna Austria

4. Department of Medicine III and Karl Landsteiner Institute for Metabolic Diseases and Nephrology Clinic Hietzing Vienna Austria

5. Joint Microbiome Facility of the Medical University of Vienna and the University of Vienna Vienna Austria

6. Department of Microbiology and Ecosystem Science, Centre for Microbiology and Environmental Systems Science University of Vienna Vienna Austria

Funder

Austrian Science Fund

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/all.15989

Reference6 articles.

1. Obesity

2. Treatment strategies for allergy and asthma

3. Obesity and Sex Affect the Immune Responses to Tick-Borne Encephalitis Booster Vaccination

4. Gut Microbiome Composition in Obese and Non-Obese Persons: A Systematic Review and Meta-Analysis

5. Th Type 1-Stimulating Activity of Lung Macrophages Inhibits Th2-Mediated Allergic Airway Inflammation by an IFN-γ-Dependent Mechanism