Tau phosphorylation and tau mislocalization mediate soluble Aβ oligomer-induced AMPA glutamate receptor signaling deficits-Reference-Cited by-同舟云学术

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Tau phosphorylation and tau mislocalization mediate soluble Aβ oligomer-induced AMPA glutamate receptor signaling deficits

Published:2014-04 Issue:7 Volume:39 Page:1214-1224
ISSN:0953-816X
Container-title:European Journal of Neuroscience
language:en
Short-container-title:Eur J Neurosci

Author:

Miller Eric C.¹²³,Teravskis Peter J.¹³⁴,Dummer Benjamin W.¹³⁴,Zhao Xiaohui³⁵,Huganir Richard L.⁶,Liao Dezhi¹²³

Affiliation:

1. Department of Neuroscience; University of Minnesota; 2101 6th St. SE Minneapolis MN 55455 USA

2. Graduate Program in Neuroscience; University of Minnesota; Minneapolis MN USA

3. N. Bud Grossman Center for Memory Research and Care; University of Minnesota; Minneapolis MN USA

4. College of Biological Sciences; University of Minnesota; Minneapolis MN USA

5. Department of Neurology; University of Minnesota; Minneapolis MN USA

6. The Solomon H. Snyder Department of Neuroscience; Johns Hopkins University School of Medicine; Baltimore MD USA

Publisher

Wiley

Subject

General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/ejn.12507/fullpdf

Reference81 articles.

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2. Role of abnormally phosphorylated tau in the breakdown of microtubules in Alzheimer disease;Alonso;Proc. Natl. Acad. Sci. USA,1994

3. Role of tau protein in both physiological and pathological conditions;Avila;Physiol. Rev.,2004

4. Neurofibrillary tangles mediate the association of amyloid load with clinical Alzheimer disease and level of cognitive function;Bennett;Arch. Neurol.,2004

5. Neuropathology of older persons without cognitive impairment from two community-based studies;Bennett;Neurology,2006

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