IL‐17A is a common and critical driver of impaired lung function and immunopathology induced by influenza virus, rhinovirus and respiratory syncytial virus-Reference-Cited by-同舟云学术

IL‐17A is a common and critical driver of impaired lung function and immunopathology induced by influenza virus, rhinovirus and respiratory syncytial virus

Published:2021-09 Issue:11 Volume:26 Page:1049-1059
ISSN:1323-7799
Container-title:Respirology
language:en
Short-container-title:Respirology

Author:

Liu Xiaoming¹²^ORCID,Nguyen Thi Hiep¹²,Sokulsky Leon¹,Li Xiang¹²^ORCID,Garcia Netto Keilah¹²^ORCID,Hsu Alan Chen‐Yu²³⁴^ORCID,Liu Chi⁵^ORCID,Laurie Karen⁶,Barr Ian⁶^ORCID,Tay Hock¹²,Eyers Fiona¹²^ORCID,Foster Paul S.¹²,Yang Ming¹²

Affiliation:

1. School of Biomedical Sciences and Pharmacy, College of Health, Medicine and Wellbeing University of Newcastle Callaghan New South Wales Australia

2. Priority Research Centre for Healthy Lungs, Hunter Medical Research Institute (HMRI) University of Newcastle New Lambton Heights New South Wales Australia

3. School of Medicine and Public Health, College of Health, Medicine and Wellbeing University of Newcastle Callaghan New South Wales Australia

4. Programme in Emerging Infectious Diseases Duke – National University of Singapore (NUS) Medical School Singapore

5. Department of Physiology, School of Basic Medicine Science Central South University Changsha China

6. WHO Collaborating Centre for Reference and Research on Influenza The Peter Doherty Institute for Infection and Immunity Melbourne Victoria Australia

Publisher

Wiley

Subject

Pulmonary and Respiratory Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/resp.14141

Reference43 articles.

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2. Respiratory Viral Infections in Infants: Causes, Clinical Symptoms, Virology, and Immunology

3. Infections and Airway Inflammation in Chronic Obstructive Pulmonary Disease Severe Exacerbations

4. Targeting IL-1β and IL-17A Driven Inflammation during Influenza-Induced Exacerbations of Chronic Lung Inflammation

5. Rate and influence of respiratory virus co-infection on pandemic (H1N1) influenza disease

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