Postmitotic neurons develop a p21‐dependent senescence‐like phenotype driven by a DNA damage response-Reference-Cited by-同舟云学术

Postmitotic neurons develop a p21‐dependent senescence‐like phenotype driven by a DNA damage response

Published:2012-09-12 Issue:6 Volume:11 Page:996-1004
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Jurk Diana¹,Wang Chunfang²,Miwa Satomi¹,Maddick Mandy¹,Korolchuk Viktor¹,Tsolou Avgi³,Gonos Efstathios S.³,Thrasivoulou Christopher⁴,Jill Saffrey M.²,Cameron Kerry¹,von Zglinicki Thomas¹

Affiliation:

1. Institute for Ageing and Health, Newcastle University, Newcastle upon Tyne NE4 5PL, UK

2. Department of Life Sciences, The Open University, Milton Keynes MK7 6AA, UK

3. National Hellenic Research Foundation, Institute of Biological Research and Biotechnology, Athens, Greece

4. Department of Anatomy and Developmental Biology, University College London, London WC1E 6DE, UK

Publisher

Wiley

Subject

Cell Biology,Ageing

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/j.1474-9726.2012.00870.x

Reference49 articles.

1. Opposing roles for p16Ink4a and p19Arf in senescence and ageing caused by BubR1 insufficiency

2. Clearance of p16Ink4a-positive senescent cells delays ageing-associated disorders

3. p53 deletion impairs clearance of chromosomal-instable stem cells in aging telomere-dysfunctional mice

4. Gross energy metabolism in mice under late onset, short term caloric restriction

5. Cellular senescence: putting the paradoxes in perspective

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