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Endoplasmic reticulum stress activates telomerase

Published:2013-10-22 Issue:1 Volume:13 Page:197-200
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Zhou Junzhi¹²,Mao Beibei³,Zhou Qi¹,Ding Deqiang¹²,Wang Miao¹,Guo Peng¹,Gao Yuhao³,Shay Jerry W.⁴,Yuan Zengqiang³,Cong Yu-Sheng¹

Affiliation:

1. Institute of Aging Research; Hangzhou Normal University School of Medicine; Hangzhou 310036 China

2. Key Laboratory for Cell Proliferation and Regulation Biology of Ministry of Education; Beijing Normal University; Beijing 100875 China

3. Institute of Biophysics; Chinese Academy of Sciences; Beijing 100101 China

4. Department of Cell Biology; UT Southwestern Medical Center; Dallas TX 75390-9039 USA

Publisher

Wiley

Subject

Cell Biology,Ageing

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/acel.12161/fullpdf

Reference11 articles.

1. Telomeres, telomerase, and human disease;Artandi;N. Engl. J. Med.,2006

2. Telomere states and cell fates;Blackburn;Nature,2000

3. Actions of human telomerase beyond telomeres;Cong;Cell Res.,2008

4. The catalytic subunit of telomerase is expressed in developing brain neurons and serves a cell survival-promoting function;Fu;J. Mol. Neurosci.,2000

5. Endoplasmic reticulum stress stimulates the expression of cyclooxygenase-2 through activation of NF-kappaB and pp38 mitogen-activated protein kinase;Hung;J. Biol. Chem.,2004

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