A novel functional polymorphism of GSTM 3 reduces clear cell renal cell carcinoma risk through enhancing its expression by interfering miR‐556 binding-Reference-Cited by-全球学者库

A novel functional polymorphism of GSTM 3 reduces clear cell renal cell carcinoma risk through enhancing its expression by interfering miR‐556 binding

Published:2018-03-22 Issue:6 Volume:22 Page:3005-3015
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J Cellular Molecular Medi

Author:

Wang Ying¹^ORCID,Yang Zi‐Ying¹,Chen Yi‐Huan¹,Li Feng²,Shen Han¹,Yu You¹,Huang Hao‐Yue¹,Shen Zhen‐Ya¹

Affiliation:

1. Department of Cardiovascular Surgery First Affiliated Hospital & Institute for Cardiovascular Science Soochow University Suzhou Jiangsu China

2. Department of Urinary Surgery First Affiliated Hospital of Soochow University Suzhou Jiangsu China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.13528

Reference19 articles.

1. Epidemiology and risk factors for kidney cancer

2. Phase I Study of Dovitinib (TKI258), an Oral FGFR, VEGFR, and PDGFR Inhibitor, in Advanced or Metastatic Renal Cell Carcinoma

3. Genome-wide association study of renal cell carcinoma identifies two susceptibility loci on 2p21 and 11q13.3

4. Common genetic variants at the 11q13.3 renal cancer susceptibility locus influence binding of HIF to an enhancer of cyclin D1 expression

5. A genome-wide association study identifies a novel susceptibility locus for renal cell carcinoma on 12p11.23

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