AMPK activation protects cells from oxidative stress‐induced senescence via autophagic flux restoration and intracellular NAD + elevation-Reference-Cited by-全球学者库

AMPK activation protects cells from oxidative stress‐induced senescence via autophagic flux restoration and intracellular NAD + elevation

Published:2016-02-18 Issue:3 Volume:15 Page:416-427
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Han Xiaojuan¹,Tai Haoran¹,Wang Xiaobo¹,Wang Zhe¹,Zhou Jiao¹,Wei Xiawei¹,Ding Yi¹,Gong Hui¹,Mo Chunfen¹,Zhang Jie¹,Qin Jianqiong¹,Ma Yuanji²,Huang Ning¹,Xiang Rong³,Xiao Hengyi¹

Affiliation:

1. Lab for Aging Research State Key Laboratory of Biotherapy and Cancer Center West China Hospital Sichuan University and Collaborative Innovation Center for Biotherapy Chengdu China

2. Center of Infectious Diseases West China Hospital Sichuan University Chengdu China

3. Department of Clinical Medicine Medical School of Nankai University Tianjin China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Aging

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/acel.12446

Reference37 articles.

1. The AMP‐ activated protein kinase AAK‐2 links energy levels and insulin ‐like signals to lifespan in C. elegans;Apfeld J;Genes Dev.,2003

2. NAD depletion by FK866 induces autophagy

3. Cell senescence and hypermitogenic arrest

4. AMP-activated protein kinase regulates nicotinamide phosphoribosyl transferase expression in skeletal muscle

5. AMPK at the Nexus of Energetics and Aging

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