Chronic Ethanol Metabolism Inhibits Hepatic Mitochondrial Superoxide Dismutase via Lysine Acetylation

Author:

Assiri Mohammed A.1ORCID,Roy Samantha R.1,Harris Peter S.1,Ali Hadi1,Liang Yongliang2,Shearn Colin T.1,Orlicky David J.3,Roede James R.1,Hirschey Matthew D.45,Backos Donald S.1,Fritz Kristofer S.1

Affiliation:

1. Skaggs School of Pharmacy and Pharmaceutical Sciences; University of Colorado, Anschutz Medical Campus; Aurora Colorado

2. Clinical Biomarkers Laboratory; Department of Medicine; Emory University; Atlanta Georgia

3. Department of Pathology; University of Colorado, Anschutz Medical Campus; Aurora Colorado

4. Department of Medicine; Division of Endocrinology, Metabolism, and Nutrition; Duke University Medical Center; Durham North Carolina

5. Department of Pharmacology and Cancer Biology; Duke University Medical Center; Durham North Carolina

Funder

University of Colorado Computational Chemistry and Biology Core Facility

NIH/NCATS

NIH/NCRR

University of Colorado Cancer Center Grant

NIH/NIAAA

King Saud University

Publisher

Wiley

Subject

Psychiatry and Mental health,Toxicology,Medicine (miscellaneous)

Reference44 articles.

1. The redox regulation of intermediary metabolism by a superoxide-aconitase rheostat;Armstrong;BioEssays,2004

2. Mechanisms and dynamics of protein acetylation in mitochondria;Baeza;Trends Biochem Sci,2016

3. CDC 2013 Alcohol and Public Health: Alcohol-Related Disease Impact (ARDI). Average for United States 2006-2010 Alcohol-Attributable Deaths Due to Excessive Alcohol Use Centers for Disease Control and Prevention (CDC) Atlanta, GA

4. Alcohol metabolism;Cederbaum;Clin Liver Dis,2012

5. Role of oxidative stress in alcohol-induced liver injury;Cederbaum;Arch Toxicol,2009

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