A role of Heat Shock Protein 70 in Photoreceptor Cell Death: Potential as a Novel Therapeutic Target in Retinal Degeneration-Reference-Cited by-同舟云学术

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A role of Heat Shock Protein 70 in Photoreceptor Cell Death: Potential as a Novel Therapeutic Target in Retinal Degeneration

Published:2015-10-28 Issue:1 Volume:22 Page:7-14
ISSN:1755-5930
Container-title:CNS Neuroscience & Therapeutics
language:en
Short-container-title:CNS Neurosci Ther

Author:

Furukawa Ayako¹,Koriyama Yoshiki¹

Affiliation:

1. Graduate School and Faculty of Pharmaceutical Sciences; Suzuka University of Medical Science; Suzuka Japan

Publisher

Wiley

Subject

Pharmacology (medical),Physiology (medical),Psychiatry and Mental health,Pharmacology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/cns.12471/fullpdf

Reference135 articles.

1. Pharmacological approaches to retinitis pigmentosa: A laboratory perspective;Guadagni;Prog Retin Eye Res,2015

2. Animal models and different therapies for treatment of retinitis pigmentosa;Rivas;Histol Histopathol,2009

3. Gene therapy in animal models of autosomal dominant retinitis pigmentosa;Rossmiller;Mol Vis,2012

4. Intraocular gene transfer of ciliary neurotrophic factor prevents death and increases responsiveness of rod photoreceptors in the retinal degeneration slow mouse;Cayouette;J Neurosci,1998

5. Retinal degeneration in the rd mouse is caused by a defect in the beta subunit of rod cGMP-phosphodiesterase;Bowes;Nature,1990

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