Recurrent RARG trinary fusion and ligand binding domain truncation in variant acute promyelocytic leukemia resistant to retinoic acid but sensitive to homoharringtonine‐based therapy
Author:
Affiliation:
1. Division of Pathology & Laboratory Medicine Hebei Yanda Lu Daopei Hospital Langfang China
2. Department of Pediatrics Peking University People's Hospital Beijing China
3. Beijing Lu Daopei Institute of Hematology Beijing China
Publisher
Wiley
Subject
Biochemistry (medical),Clinical Biochemistry,Hematology,General Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/ijlh.14150
Reference10 articles.
1. A global study for acute myeloid leukemia with RARG rearrangement
2. Critical role of retinoid/rexinoid signaling in mediating transformation and therapeutic response of NUP98-RARG leukemia
3. Arsenic Combined With All-Trans Retinoic Acid for Pediatric Acute Promyelocytic Leukemia: Report From the CCLG-APL2016 Protocol Study
4. A short report of novel RARG-HNRNPM fusion gene in resembling acute promyelocytic leukemia
5. A novel <i>SART3::RARG</i> fusion gene in acute myeloid leukemia with acute promyelocytic leukemia phenotype and differentiation escape to retinoic acid
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