Mitochondrial function and energy metabolism in neuronal HT22 cells resistant to oxidative stress-Reference-Cited by-同舟云学术

Mitochondrial function and energy metabolism in neuronal HT22 cells resistant to oxidative stress

Published:2014-03-28 Issue:8 Volume:171 Page:2147-2158
ISSN:0007-1188
Container-title:British Journal of Pharmacology
language:en
Short-container-title:Br J Pharmacol

Author:

Pfeiffer Annika¹,Jaeckel Martin²,Lewerenz Jan³,Noack Rebecca¹,Pouya Alireza¹,Schacht Teresa¹,Hoffmann Christina¹,Winter Jennifer⁴,Schweiger Susann⁴,Schäfer Michael K E²,Methner Axel¹

Affiliation:

1. Department of Neurology; University Medical Center and Focus Program Translational Neuroscience (FTN) of the Johannes Gutenberg-University Mainz; Mainz Germany

2. Department of Anesthesiology; University Medical Center and Focus Program Translational Neuroscience (FTN) of the Johannes Gutenberg-University Mainz; Mainz Germany

3. Department of Neurology; University of Ulm; Ulm Germany

4. Institute of Human Genetics; University Medical Center and Focus Program Translational Neuroscience (FTN) of the Johannes Gutenberg-University Mainz; Mainz Germany

Funder

Else Krï¿½ner-Fresenius Stiftung

Publisher

Wiley

Subject

Pharmacology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/bph.12549/fullpdf

Reference37 articles.

1. Mechanisms of oxidative glutamate toxicity: the glutamate/cystine antiporter system xc− as a neuroprotective drug target;Albrecht;CNS Neurol Disord Drug Targets,2010

2. Effects of dimethyl fumarate on neuroprotection and immunomodulation;Albrecht;J Neuroinflammation,2012

3. The concise guide to pharmacology 2013/2014. Transporters;Alexander;Br J Pharmacol,2013a