A decade of progress in adipose tissue macrophage biology

Author:

Hill Andrea A.1,Reid Bolus W.1,Hasty Alyssa H.1

Affiliation:

1. Department of Molecular Physiology and Biophysics; Vanderbilt University School of Medicine; Nashville TN USA

Funder

American Heart Association

National Institute of Diabetes and Digestive and Kidney Diseases

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Reference143 articles.

1. Model for cachexia in chronic disease: secretory products of endotoxin-stimulated macrophages induce a catabolic state in 3T3-L1 adipocytes;Pekala;Trans Assoc Am Physicians,1984

2. A macrophage factor inhibits adipocyte gene expression: an in vitro model of cachexia;Torti;Science,1985

3. Identity of tumour necrosis factor and the macrophage-secreted factor cachectin;Beutler;Nature,1985

4. Regulation of lipoprotein lipase synthesis in 3T3-L1 adipocytes by cachectin. Further proof for identity with tumour necrosis factor;Price;Biochem J,1986

5. Critical nodes in signalling pathways: insights into insulin action;Taniguchi;Nat Rev Mol Cell Biol,2006

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