Islet‐specific <scp>CD8</scp> <sup>+</sup> T cells gain effector function in the gut lymphoid tissues <i>via</i> bystander activation not molecular mimicry-Reference-Cited by-同舟云学术

Islet‐specific CD8 ⁺ T cells gain effector function in the gut lymphoid tissues via bystander activation not molecular mimicry

Published:2022-11 Issue:1 Volume:101 Page:36-48
ISSN:0818-9641
Container-title:Immunology & Cell Biology
language:en
Short-container-title:Immunol Cell Biol

Author:

Okada Mirei¹^ORCID,Zhang Vivian¹,Loaiza Naranjo Jeniffer D¹,Tillett Bree J¹,Wong F Susan²,Steptoe Raymond J¹,Bergot Anne‐Sophie¹,Hamilton‐Williams Emma E¹^ORCID

Affiliation:

1. The University of Queensland Diamantina Institute, The University of Queensland Woolloongabba QLDAustralia

2. Division of Infection and Immunity and Systems Immunity University Research Institute, School of Medicine Cardiff University Cardiff UK

Funder

Juvenile Diabetes Research Foundation International

Fondazione Diabete Ricerca

Children's Hospital Foundation

Publisher

Wiley

Subject

Cell Biology,Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/imcb.12593

Reference50 articles.

1. Environmental risk factors for type 1 diabetes

2. Microbiome and type 1 diabetes

3. Variation in Microbiome LPS Immunogenicity Contributes to Autoimmunity in Humans

4. Loss of gut barrier integrity triggers activation of islet-reactive T cells and autoimmune diabetes

5. Gut microbial metabolites limit the frequency of autoimmune T cells and protect against type 1 diabetes

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