Two Transcription Factors, E1AF and N-myc, Correlate with the Invasiveness of Neuroblastoma Cell Lines
Author:
Publisher
Wiley
Subject
Cancer Research,Oncology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1349-7006.1997.tb00395.x/fullpdf
Reference51 articles.
1. Transposition and amplification of oncogene-related sequences in human neuroblastomas;Kohl;Cell,1983
2. Amplification of N-myc in untreated human neuroblastomas correlates with advanced disease stage;Brodeur;Science,1984
3. Embryonic lethality in mice homozygous for a targeted disruption of the N-myc gene;Charron;Genes Dev.,1992
4. Loss of N-myc function results in embryonic lethality and failure of the epithelial component of the embryo to develop;Stanton;Genes Dev.,1992
5. Surgical aspects of N-myc oncogene amplification of neuroblastoma;Nakagawara;Surgery,1988
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1. Acquired resistance to oxaliplatin is not directly associated with increased resistance to DNA damage in SK-N-ASrOXALI4000, a newly established oxaliplatin-resistant sub-line of the neuroblastoma cell line SK-N-AS;PLOS ONE;2017-02-13
2. Requirement of PEA3 for Transcriptional Activation of FAK Gene in Tumor Metastasis;PLoS ONE;2013-11-18
3. Identification of E1AF as a Target Gene of E2F1-induced Apoptosis in Response to DNA Damage;Journal of Biochemistry;2008-06-16
4. Heterogeneity in retinoic acid signaling in neuroblastomas: Role of matrix metalloproteinases in retinoic acid-induced differentiation;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2007-09
5. ERM/ETV5 Up-regulation Plays a Role during Myometrial Infiltration through Matrix Metalloproteinase-2 Activation in Endometrial Cancer;Cancer Research;2007-07-15
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