Prevention by (±)-8-hydroxy-2-(di-npropylamino)tetralin of both catalepsy and the rises in rat striatal dopamine metabolism caused by haloperidol

Author:

Andersen Heidi L.,Kilpatrick Ian C.

Publisher

Wiley

Subject

Pharmacology

Reference62 articles.

1. Evidence for selective inhibition of limbic forebrain dopamine synthesis by 8-OH-DPAT in the rat;AHLENIUS;Naunyn-Schmied. Arch. Pharmacol.,1989

2. Increased dopamine turnover in the ventral striatum by 8-OH-DPAT administration in the rat;AHLENIUS;J. Pharm. Pharmacol.,1990

3. Catalepsy, brain dopamine metabolism and the interaction between haloperidol and 8-OH-DPAT;ANDERSEN;Br. J. Pharmacol.,1995

4. The 5-HT1A receptor selective ligands, (R)-8-OH-DPAT and (S)-UH-301, differentially affect the activity of midbrain dopamine neurons;ARBORELIUS;Naunyn-Schmied. Arch. Pharmacol.,1993a

5. (R)-8-OH-DPAT preferentially increases dopamine release in rat medial prefrontal cortex;ARBORELIUS;Acta Physiol. Scand.,1993b

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