Angiopoietin‐like protein 4 deficiency augments liver fibrosis in liver diseases such as nonalcoholic steatohepatitis in mice through enhanced free cholesterol accumulation in hepatic stellate cells-Reference-Cited by-同舟云学术

Angiopoietin‐like protein 4 deficiency augments liver fibrosis in liver diseases such as nonalcoholic steatohepatitis in mice through enhanced free cholesterol accumulation in hepatic stellate cells

Published:2021-04-07 Issue:5 Volume:51 Page:580-592
ISSN:1386-6346
Container-title:Hepatology Research
language:en
Short-container-title:Hepatology Research

Author:

Teratani Toshiaki¹,Tomita Kengo²,Wada Akinori²,Sugihara Nao²,Higashiyama Masaaki²,Inaba Kenichi²,Horiuchi Kazuki²,Hanawa Yoshinori²,Nishii Shin²,Mizoguchi Akinori²,Tanemoto Rina²,Ito Suguru²,Okada Yoshikiyo²,Kurihara Chie²,Akita Yoshihiro²,Narimatsu Kazuyuki²,Watanabe Chikako²,Komoto Shunsuke²,Oike Yuichi³,Miura Soichiro²⁴,Hokari Ryota²,Kanai Takanori¹

Affiliation:

1. Division of Gastroenterology and Hepatology Department of Internal Medicine Keio University School of Medicine Shinjuku‐ku Tokyo Japan

2. Division of Gastroenterology and Hepatology Department of Internal Medicine National Defense Medical College Tokorozawa‐shi Saitama Japan

3. Department of Molecular Genetics Graduate School of Medical Sciences Kumamoto University Chuo‐ku Kumamoto Japan

4. International University of Health and Welfare Graduate School Minato‐ku Tokyo Japan

Publisher

Wiley

Subject

Infectious Diseases,Hepatology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/hepr.13603

Reference32 articles.

1. Free cholesterol accumulation in hepatic stellate cells: Mechanism of liver fibrosis aggravation in nonalcoholic steatohepatitis in mice

2. Lipoprotein Lipase Up‐regulation in Hepatic Stellate Cells Exacerbates Liver Fibrosis in Nonalcoholic Steatohepatitis in Mice

3. Angiopoietin-like protein 4 is a potent hyperlipidemia-inducing factor in mice and inhibitor of lipoprotein lipase

4. The Role of Angiopoietin-Like Proteins in Angiogenesis and Metabolism

5. Not all angiopoietin-like proteins (Angptls) are created equal: insights from molecular, genetic, and pharmacological studies on the mechanism of LPL inhibition by Angptl3 and Angptl4

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