Elevation of androgen receptor promotes prostate cancer metastasis by induction of epithelial‐mesenchymal transition and reduction of KAT 5-Reference-Cited by-全球学者库

Elevation of androgen receptor promotes prostate cancer metastasis by induction of epithelial‐mesenchymal transition and reduction of KAT 5

Published:2018-09-25 Issue:11 Volume:109 Page:3564-3574
ISSN:1347-9032
Container-title:Cancer Science
language:en
Short-container-title:Cancer Sci

Author:

Lin Ching‐Yu¹,Jan Yee‐Jee²³,Kuo Li‐Kuo⁴⁵,Wang Bi‐Juan¹,Huo Chieh¹,Jiang Shih Sheng⁶,Chen Shyh‐Chang²,Kuo Ying‐Yu¹⁷,Chang Chuang‐Rung⁷,Chuu Chih‐Pin¹⁸⁹¹⁰^ORCID

Affiliation:

1. Institute of Cellular and System Medicine National Health Research Institutes Miaoli Taiwan

2. Department of Pathology and Laboratory Medicine Taichung Veterans General Hospital Taichung Taiwan

3. Medical College of Chung Shan Medical University Taichung Taiwan

4. Division of Pulmonary and Critical Care Medicine Department of Internal Medicine Mackay Memorial Hospital Taipei City Taiwan

5. Department of Nursing Mackay Medical College Taipei City Taiwan

6. Nation Institute of Cancer Research National Health Research Institutes Miaoli Taiwan

7. Institute of Biotechnology National Tsing Hua University Hsinchu City Taiwan

8. Graduate Program for Aging China Medical University Taichung City Taiwan

9. Graduate Institute of Basic Medical Science China Medical University Taichung City Taiwan

10. Biotechnology Center National Chung Hsing University Taichung City Taiwan

Funder

National Health Research Institutes

Ministry of Science and Technology

Publisher

Wiley

Subject

Cancer Research,Oncology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/cas.13776

Reference40 articles.

1. Selective Retention of Dihydrotestosterone by Prostatic Nuclei

2. Molecular Action of Androgen in the Normal and Neoplastic Prostate

3. Inhibition of steroid 5α-reductase by specific aliphatic unsaturated fatty acids

4. The development of androgen-independent prostate cancer

5. Androgen Causes Growth Suppression and Reversion of Androgen-Independent Prostate Cancer Xenografts to an Androgen-Stimulated Phenotype in Athymic Mice

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