Nrf2 stabilization prevents critical oxidative damage in Down syndrome cells-Reference-Cited by-同舟云学术

Nrf2 stabilization prevents critical oxidative damage in Down syndrome cells

Published:2018-07-20 Issue:5 Volume:17 Page:e12812
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Zamponi Emiliano¹,Zamponi Nahuel²,Coskun Pinar³,Quassollo Gonzalo¹,Lorenzo Alfredo¹,Cannas Sergio A.⁴,Pigino Gustavo¹,Chialvo Dante R.⁵,Gardiner Katheleen⁶,Busciglio Jorge³,Helguera Pablo¹^ORCID

Affiliation:

1. Instituto de Investigación Médica Mercedes y Martín Ferreyra; INIMEC-CONICET-Universidad Nacional de Córdoba; Cordoba Argentina

2. Department of Medicine, Division of Hematology and Medical Oncology; Weill Cornell Medicine; New York New York

3. Department of Neurobiology and Behavior, Institute for Memory Impairments and Neurological Disorders and Center for the Neurobiology of Learning and Memory; University of California Irvine; Irvine California

4. Instituto de Física Enrique Gaviola (IFEG-CONICET); FAMAFyC, UNC; Cordoba Argentina

5. Center for Complex Systems and Brain Sciences (CEMSC3); UNSAM; San Martin Argentina

6. Department of Pediatrics, Linda Crnic Institute for Down Syndrome; University of Colorado Denver School of Medicine; Aurora Colorado

Funder

Fondo Nacional de Ciencia y Técnica

National Institutes of Health Alzheimer's Disease Research Center Grant

ADRC-UCI pilot project

International Union of Biochemistry and Molecular Biology

Publisher

Wiley

Subject

Cell Biology,Ageing

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/acel.12812/fullpdf

Reference55 articles.

1. Caspase-3-dependent proteolytic cleavage of protein kinase Cdelta is essential for oxidative stress-mediated dopaminergic cell death after exposure to methylcyclopentadienyl manganese tricarbonyl;Anantharam;Journal of Neuroscience,2002

2. Lipid peroxidation and superoxide dismutase-1 and glutathione peroxidase activities in trisomy 16 fetal mice and human trisomy 21 fibroblasts;Annerén;Pediatric Research,1987

3. Chromosome 21 and down syndrome: From genomics to pathophysiology;Antonarakis;Nature Reviews Genetics,2004

4. Overexpression of catalase in cytosolic or mitochondrial compartment protects HepG2 cells against oxidative injury;Bai;Journal of Biological Chemistry,1999

5. Mitochondria, oxidants, and aging;Balaban;Cell,2005

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