Functional abnormalities in iPSC‐derived cardiomyocytes generated from CPVT1 and CPVT2 patients carrying ryanodine or calsequestrin mutations-Reference-Cited by-同舟云学术

Functional abnormalities in iPSC‐derived cardiomyocytes generated from CPVT1 and CPVT2 patients carrying ryanodine or calsequestrin mutations

Published:2015-07-08 Issue:8 Volume:19 Page:2006-2018
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J. Cell. Mol. Med.

Author:

Novak Atara¹²³,Barad Lili¹²³,Lorber Avraham³⁴,Gherghiceanu Mihaela⁵,Reiter Irina¹²³,Eisen Binyamin¹²³,Eldor Liron⁶,Itskovitz‐Eldor Joseph²³,Eldar Michael⁷,Arad Michael⁷,Binah Ofer¹²³

Affiliation:

1. Department of Physiology Technion Haifa Israel

2. The Rappaport Institute for Research in the Medical Sciences Technion Haifa Israel

3. Ruth & Bruce Rappaport Faculty of Medicine Technion Haifa Israel

4. Department of Pediatric Cardiology Rambam Health Care Campus Haifa Israel

5. ‘Victor Babes’ National Institute of Pathology Bucharest Romania

6. Department of Plastic Surgery Rambam Health Care Campus Haifa Israel

7. Leviev Heart Center Sheba Medical Center Tel Hashomer and Sackler School of Medicine Tel Aviv University Tel Aviv Israel

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.12581

Reference40 articles.

1. Catecholaminergic polymorphic ventricular tachycardia: Recent mechanistic insights

2. Disruption of calcium homeostasis and arrhythmogenesis induced by mutations in the cardiac ryanodine receptor and calsequestrin

3. Inherited Dysfunction of Sarcoplasmic Reticulum Ca2+Handling and Arrhythmogenesis

4. In vitro Modeling of Ryanodine Receptor 2 Dysfunction Using Human Induced Pluripotent Stem Cells

5. Modeling of Catecholaminergic Polymorphic Ventricular Tachycardia With Patient-Specific Human-Induced Pluripotent Stem Cells

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