Association between Thromboembolic Events and the JAK2 V617F Mutation in Myeloproliferative Neoplasms
Author:
Affiliation:
1. Department of Medicine, Kurume University School of Medicine
Publisher
Kurume Medical Journal
Subject
General Medicine
Link
https://www.jstage.jst.go.jp/article/kurumemedj/60/3.4/60_MS63001/_pdf
Reference36 articles.
1. 1. Kralovics R, Passamonti F, Buser AS, Teo SS, Skoda RC et al. A Gain-of-Function mutation of JAK2 in myeloproliferative disorders. N Engl J Med 2005; 352:1779-90.
2. 2. Baxter E, Scott LM, Campbell P, East C, Fourouclas N et al. Acquired mutation of the tyrosine kinase JAK2 in human myeloproliferative disorders. Lancet 2005; 365:1054-61.
3. 3. James C, Ugo V, Casadevall N, Constantinescu SN, Vainchenker W. A JAK2 mutation in myeloproliferative disorders: pathogenesis and therapeutic and scientific prospects. Trends in Molecular Medicine 2005; 11:1471-4914.
4. 4. Vainchenker W, Delhommeau F, Constantinescu SN, Bernard OA. New mutations and pathogenesis of myeloproliferative neoplasms. Blood 2011; 118(7):1723-1735.
5. 5. Levine RL, Wernig G. Role of JAK-STAT Signaling in the Pathogenesis of myeloproliferative disorders. Hematology 2006; 233-239.
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