Early-onset Amyloid Deposition and Cognitive Deficits in Transgenic Mice Expressing a Double Mutant Form of Amyloid Precursor Protein 695-Reference-Cited by-同舟云学术

Early-onset Amyloid Deposition and Cognitive Deficits in Transgenic Mice Expressing a Double Mutant Form of Amyloid Precursor Protein 695

Published:2001-06 Issue:24 Volume:276 Page:21562-21570
ISSN:0021-9258
Container-title:Journal of Biological Chemistry
language:en
Short-container-title:Journal of Biological Chemistry

Author:

Chishti M.Azhar,Yang Dun-Shen,Janus Christopher,Phinney Amie L.,Horne Patrick,Pearson Jacqueline,Strome Robert,Zuker Noah,Loukides James,French Janet,Turner Sherry,Lozza Gianluca,Grilli Mariagrazia,Kunicki Suzanne,Morissette Céline,Paquette Julie,Gervais Francine,Bergeron Catherine,Fraser Paul E.,Carlson George A.,George-Hyslop Peter St.,Westaway David

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference73 articles.

1. Aβ peptide immunization reduces behavioural impairment and plaques in a model of Alzheimer's disease

2. Mutation of the β-amyloid precursor protein in familial Alzheimer's disease increases β-protein production

3. Structural alterations in the peptide backbone of beta-amyloid core protein may account for its deposition and stability in Alzheimer's disease.

4. Increased β-amyloid release and levels of amyloid precursor protein (APP) in fibroblast cell lines from family members with the Swedish Alzheimer's disease APP670/671 mutation

5. An Increased Percentage of Long Amyloid β Protein Secreted by Familial Amyloid β Protein Precursor (βApp 717 ) Mutants

Cited by 806 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Transgenic amyloid precursor protein mouse models of amyloidosis. Incomplete models for Alzheimer's disease but effective predictors of anti‐amyloid therapies;Alzheimer's & Dementia;2023-12-12

2. A C1qTNF3 collagen domain fusion chaperones diverse secreted proteins and anti-Aβ scFvs: Applications for gene therapies;Molecular Therapy - Methods & Clinical Development;2023-12

3. Aβ Amyloid Scaffolds the Accumulation of Matrisome and Additional Proteins in Alzheimer’s Disease;2023-12-01

4. Enhanced prefrontal nicotinic signaling as evidence of active compensation in Alzheimer’s disease models;2023-11-13

5. P2X7R influences tau aggregate burden in human tauopathies and shows distinct signalling in microglia and astrocytes;Brain, Behavior, and Immunity;2023-11