Insulin-like Growth Factor I Increases αVβ3 Affinity by Increasing the Amount of Integrin-associated Protein That Is Associated with Non-raft Domains of the Cellular Membrane
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference24 articles.
1. Interaction of circulating cell-derived and plasma growth factors in stimulating cultured smooth muscle cell replication
2. Glycosylation of Insulin-Like Growth Factor Binding Protein-3 (IGFBP-3) is not Required for Potentiation IGF-I Action: Evidence for Processing of Cell-Bound IGFBP-3*
3. Insulin-like growth factor-I and platelet-derived growth factor-BB induce directed migration of human arterial smooth muscle cells via signaling pathways that are distinct from those of proliferation.
4. Synthetic αVβ3 Antagonists Inhibit Insulin-Like Growth Factor-I-Stimulated Smooth Muscle Cell Migration and Replication1
5. Ligand occupancy of the alpha-V-beta3 integrin is necessary for smooth muscle cells to migrate in response to insulin-like growth factor.
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1. Something old, something new and something borrowed: emerging paradigm of insulin-like growth factor type 1 receptor (IGF-1R) signaling regulation;Cellular and Molecular Life Sciences;2013-11-26
2. Dietary n-3 polyunsaturated fatty acid supplementation alters the expression of genes involved in the control of fertility in the bovine uterine endometrium;Physiological Genomics;2012-09-15
3. Syndecan-1 couples the insulin-like growth factor-1 receptor to inside-out integrin activation;Journal of Cell Science;2010-11-01
4. Cross-talk between the insulin-like growth factor (IGF) axis and membrane integrins to regulate cell physiology;Journal of Cellular Physiology;2010-04-28
5. Regulation of Cross-linked Actin Network (CLAN) Formation in Human Trabecular Meshwork (HTM) Cells by Convergence of Distinct β1 and β3 Integrin Pathways;Investigative Opthalmology & Visual Science;2009-12-01
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