Latent Membrane Protein 1 of Epstein-Barr Virus Stimulates Processing of NF-κB2 p100 to p52
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference69 articles.
1. Alternate replication in B cells and epithelial cells switches tropism of Epstein–Barr virus
2. Epstein-Barr virus: exploiting the immune system
3. The Epstein-Barr virus LMP1 cytoplasmic carboxy terminus is essential for B-lymphocyte transformation; fibroblast cocultivation complements a critical function within the terminal 155 residues
4. Inhibition of in vitro proliferation of Epstein Barr Virus infected B cells by an antisense oligodeoxynucleotide targeted against EBV latent membrane protein LMP1
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2. Viral-Targeted Strategies Against EBV-Associated Lymphoproliferative Diseases;Frontiers in Oncology;2019-02-26
3. CADM1 is essential for KSHV-encoded vGPCR-and vFLIP-mediated chronic NF-κB activation;PLOS Pathogens;2018-04-26
4. NF-κB Inhibitors in Head and Neck Cancer;Letters in Drug Design & Discovery;2017-04-06
5. The Latent Membrane Protein 1 (LMP1);Epstein Barr Virus Volume 2;2015
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