c-Jun N-terminal Kinase (JNK)-dependent Acute Liver Injury from Acetaminophen or Tumor Necrosis Factor (TNF) Requires Mitochondrial Sab Protein Expression in Mice
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference39 articles.
1. The c-jun kinase/stress-activated pathway: Regulation, function and role in human disease
2. Cell Signaling in Oxidative Stress-Induced Liver Injury
3. Redox Regulation of Tumor Necrosis Factor Signaling
4. The JNK signal transduction pathway
5. NF-κB inhibition sensitizes hepatocytes to TNF-induced apoptosis through a sustained activation of JNK and c-Jun
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