Proliferative Defect and Embryonic Lethality in Mice Homozygous for a Deletion in the p110α Subunit of Phosphoinositide 3-Kinase
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference36 articles.
1. Cross-talk between phospholipase C and phosphoinositide 3-kinase signalling pathways
2. Phosphoinositide 3-kinase: the key switch mechanism in insulin signalling
3. Biochemical characterization of the free catalytic p110 alpha and the complexed heterodimeric p110 alpha.p85 alpha forms of the mammalian phosphatidylinositol 3-kinase.
4. Phosphatidylinositol 3-kinase
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