Silencing MR-1 attenuates atherosclerosis in ApoE−/− mice induced by angiotensin II through FAK-Akt–mTOR-NF-kappaB signaling pathway
Author:
Affiliation:
1. Hunan Environment-Biological Polytechnic College, Hengyang Hunan 421005, China.
2. Key Lab of Antibiotic Biotechnology, Institute of Medicinal Biotechnology, Chinese Academy of Medical Sciences, Beijing 100050, China.
Publisher
The Korean Physiological Society and The Korean Society of Pharmacology
Subject
Pharmacology,Physiology
Link
https://synapse.koreamed.org/pdf/10.4196/kjpp.2018.22.2.127
Reference25 articles.
1. Inhibition of PTEN expression and activity by angiotensin II induces proliferation and migration of vascular smooth muscle cells
2. MicroRNA-761 inhibits Angiotensin II-induced vascular smooth muscle cell proliferation and migration by targeting mammalian target of rapamycin
3. Angiotensin-(1–7) counteracts the effects of Ang II on vascular smooth muscle cells, vascular remodeling and hemorrhagic stroke: Role of the NFкB inflammatory pathway
4. Alpha1beta1 and integrin-linked kinase interact and modulate angiotensin II effects in vascular smooth muscle cells
5. Angiotensin-(1–7) abrogates angiotensin II-induced proliferation, migration and inflammation in VSMCs through inactivation of ROS-mediated PI3K/Akt and MAPK/ERK signaling pathways
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