Co-expression of multiple transgenes in mouse CNS: a comparison of strategies
Author:
Publisher
Elsevier BV
Subject
Molecular Biology,Bioengineering,Biotechnology
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1. Familial Alzheimer's Disease–Linked Presenilin 1 Variants Elevate Aβ1–42/1–40 Ratio In Vitro and In Vivo
2. Accelerated Amyloid Deposition in the Brains of Transgenic Mice Coexpressing Mutant Presenilin 1 and Amyloid Precursor Proteins
3. Genetic Modification of the Phenotypes Produced by Amyloid Precursor Protein Overexpression in Transgenic Mice
4. SOD1 rescues cerebral endothelial dysfunction in mice overexpressing amyloid precursor protein
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