Cardiopulmonary resuscitation ameliorates myocardial mitochondrial dysfunction in a cardiac arrest rat model

Author:

Xu WenORCID,Fu Yue,Jiang Longyuan,Yang ZhengfeiORCID,Wang Yue,Tang Wanchun,Fang Xiangshao

Funder

National Natural Science Foundation of China

Leading Talents in Pearl River Talent Plan of Guangdong Province

Guangdong Science and Technology Department

Publisher

Elsevier BV

Subject

Emergency Medicine,General Medicine

Reference33 articles.

1. Duration of untreated cardiac arrest and clinical relevance of animal experiments: the relationship between the “No-Flow” duration and the severity of post-cardiac arrest syndrome in a porcine model;Babini;Shock,2018

2. Decreased cAMP level and decreased downregulation of β1-adrenoceptor expression in therapeutic hypothermia-resuscitated myocardium are associated with improved post-resuscitation myocardial function;Wang;Journal of the American Heart Association Cardiovascular & Cerebrovascular Disease,2018

3. Echocardiographic patterns of postresuscitation myocardial dysfunction;Cha;Resuscitation,2018

4. Erratum to: European Resuscitation Council and European Society of Intensive Care Medicine 2015 guidelines for post-resuscitation care;JP;Intensive Care Med,2015

5. Biochemical dysfunction in heart mitochondria exposed to ischaemia and reperfusion;Solaini;Biochem J,2005

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