Altered myofilament structure and function in dogs with Duchenne muscular dystrophy cardiomyopathy
Author:
Funder
National Institutes of Health
Publisher
Elsevier BV
Subject
Cardiology and Cardiovascular Medicine,Molecular Biology
Reference58 articles.
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3. Bradykinin restores left ventricular function, sarcomeric protein phosphorylation, and e/nNOS levels in dogs with Duchenne muscular dystrophy cardiomyopathy;JB;Cardiovasc. Res.,2012
4. Chronic administration of membrane sealant prevents severe cardiac injury and ventricular dilatation in dystrophic dogs;Townsend;J. Clin. Invest.,2010
5. Differential contribution of cardiac sarcomeric proteins in the myofibrillar force response to stretch;Ait Mou;Pflugers Arch.,2008
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1. Ryanodine receptor dysfunction causes senescence and fibrosis in Duchenne dilated cardiomyopathy;Journal of Cachexia, Sarcopenia and Muscle;2024-01-14
2. Dystrophic cardiomyopathy: role of the cardiac myofilaments;Frontiers in Physiology;2023-06-09
3. Regulation of the evolutionarily conserved muscle myofibrillar matrix by cell type dependent and independent mechanisms;Nature Communications;2022-05-13
4. Small Angle X-ray Diffraction as a Tool for Structural Characterization of Muscle Disease;International Journal of Molecular Sciences;2022-03-11
5. Stabilizing Ryanodine Receptors Improves Left Ventricular Function in Juvenile Dogs With Duchenne Muscular Dystrophy;Journal of the American College of Cardiology;2021-12
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