Cardiac potassium channel dysfunction in sudden infant death syndrome-Reference-Cited by-同舟云学术

Cardiac potassium channel dysfunction in sudden infant death syndrome

Published:2008-03 Issue:3 Volume:44 Page:571-581
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Rhodes Troy E.,Abraham Robert L.,Welch Richard C.,Vanoye Carlos G.,Crotti Lia,Arnestad Marianne,Insolia Roberto,Pedrazzini Matteo,Ferrandi Chiara,Vege Ashild,Rognum Torleiv,Roden Dan M.,Schwartz Peter J.,George Alfred L.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference33 articles.

1. Cardiac sympathetic innervation and the sudden infant death syndrome. A possible pathogenetic link;Schwartz;Am J Med,1976

2. Prolonged QT interval and cardiac arrhythmias in two neonates: sudden infant death syndrome in one case;Southall;Arch Dis Child,1979

3. Potential role of QT interval prolongation in sudden infant death syndrome;Maron;Circulation,1976

4. Postmortem molecular analysis of SCN5A defects in sudden infant death syndrome;Ackerman;JAMA,2001

5. Sudden infant death syndrome: how significant are the cardiac channelopathies?;Tester;Cardiovasc Res,2005

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