Ablation of the cardiac ryanodine receptor phospho-site Ser2808 does not alter the adrenergic response or the progression to heart failure in mice. Elimination of the genetic background as critical variable

Author:

Alvarado Francisco J.,Chen Xi,Valdivia Héctor H.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Cited by 16 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献

1. Clustering properties of the cardiac ryanodine receptor in health and heart failure;Journal of Molecular and Cellular Cardiology;2023-12

2. Therapeutic Approaches of Ryanodine Receptor-Associated Heart Diseases;International Journal of Molecular Sciences;2022-04-18

3. RyR2 and Calcium Release in Heart Failure;Frontiers in Physiology;2021-10-08

4. An Overview of FGF-23 as a Novel Candidate Biomarker of Cardiovascular Risk;Frontiers in Physiology;2021-03-09

5. Stress-driven cardiac calcium mishandling via a kinase-to-kinase crosstalk;Pflügers Archiv - European Journal of Physiology;2021-02-15

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