Ablation of the cardiac ryanodine receptor phospho-site Ser2808 does not alter the adrenergic response or the progression to heart failure in mice. Elimination of the genetic background as critical variable
Author:
Publisher
Elsevier BV
Subject
Cardiology and Cardiovascular Medicine,Molecular Biology
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1. Ablation of three major phospho-sites in RyR2 preserves the global adrenergic response but creates an arrhythmogenic substrate;2024-07-10
2. Abnormal phosphorylation / dephosphorylation and Ca2+ dysfunction in heart failure;Heart Failure Reviews;2024-03-18
3. Clustering properties of the cardiac ryanodine receptor in health and heart failure;Journal of Molecular and Cellular Cardiology;2023-12
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